‘People with Parkinson’s disease 0.56 – could report times as likely never smoke, and 0.30 times as likely current smokers report compared unaffected relatives, ‘the authors write. ‘Increasing intensity of coffee drinking was inversely associated with Parkinson’s disease. Increasing dosage and intensity of total caffeine consumption were also inversely are connected, at high doses, the significant inverse association with Parkinson’s disease, ‘There was no association between NSAIDs and Parkinson’s disease. The biological mechanisms by which smoking and caffeine in individuals at risk of Parkinson’s disease might work is unknown, the authors state. ‘Given the complexity of Parkinson’s disease, these environmental factors likely do not exert their effects in isolation, of of gene-environment interactions in determining Parkinson’s disease susceptibility, ‘they conclude.
Hancock, who, Duke University Medical Center, and colleagues examined the relationship between smoking, caffeine and NSAID use and Parkinson’s disease in 356 Parkinson’s disease patients (average age 66 , and 0.30 and 317 family members without the disease (average age 63, participants clinically clinically to confirm their Parkinson’s disease interviewed interviewed by phone their their exposure to environmental factors.. And caffeine inversely with Parkinson’s disease – smoking people with Parkinson’s disease are less likely or consume large doses of caffeine than their family members who are not the disease, according to a report in the April issue of Archives of Neurology one of the JAMA / Archives journals.
Dana B.Asthma attacks, subtlety mutations Add immune Generated risk for asthma.
A gene responsible for of a protein to determine whether the immune cells live or die show subtle differences in in some people with asthma coded, a team of John Hopkins explorer reviews in June the led the European Journal of Human Genetics.
The investigators found in prior studies, if they bound antibodies or specifically engineered designed to sugar-coated polymers to the particular Siglec – 8 protein on eosinophils, the cell died in surprising an effect that that might be useful in controlling of an allergy and attack of asthma. Indeed, the Bochner lab is prospective in tracking to possibility of developing new on on treatments that activate Siglec-8.